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46 Meanwhile, critical illness neuropathy was been reported in 10% of intubated patients with COVID-19, with prolonged insults such as intense inflammation and nerve compressions as potential etiological agents

GLP-1 and exendin-4 transiently enhance GABAA receptor-mediated synaptic and tonic currents in rat hippocampal CA3 pyramidal neurons

[15] This is the study we'll focus on, but the other studies cited above can be read for more background