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doi:10.1111/j.1745-7599.2009.00485.x
These findings, plus the observation in which the very low level of Epac1 is detectable in pancreatic islets (Song et al., 2011), imply that Epac2A is predominant downstream effector when cAMP triggers the PKA-independent signaling in pancreatic -cells
These calcium-regulatory effects also contribute to inhibiting pulmonary vein (PV) automaticity: as a primary trigger of atrial fibrillation, PVs are modulated by GLP-1RAs, which affect PV electrical activity, ionic properties, and intracellular calcium homeostasis to suppress PV automaticity (143)
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