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Microglial activation induces nitric oxide signalling and alters protein S-nitrosylation patterns in extracellular vesicles

Growth hormone production peaks in adolescence and declines through adulthood, and IGF-1, the downstream messenger that drives muscle protein synthesis and tissue repair, declines along with it

The trend of MDA, SOD and GSH changes in MT1G overexpressing GC cell lines is consistent with the trend of MDA, SOD and GSH changes during ferroptosis

Tirzepatide acts on both GIP and GLP-1 pathways

Molecular physiology of reverse cholesterol transport