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Results Inhibition of glutathione metabolism depletes neuronal glutamate and affects excitatory transmission To test the hypothesis that glutathione is a significant reservoir for glutamate, we treated neuronal cells with molecular inhibitors targeting enzymes of the glutathione metabolic cycle: L-2-imidazolidone-4-carboxylate (2I4C), acivicin, L-buthionine sulfoximine (BSO), and sulforaphane

Helps reduce inflammation and ease chronic nerve discomfort

Determination of fraction of left ventricular volume ejected per beat and of ventricular end-diastolic and residual volumes

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Another enhancer, zonula occludens toxin (Zot), has been shown to reversibly increase the intestinal absorption of insulin and immunoglobulins

J Fisheries Mar Sci Educ