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Rice DC, Kim HW, Putnam JB, Smythe WR, Swisher SG , Vaporciyan AA, Walsh GL, Lee JJ, Sabichi A, Lippman SM, Roth JA
The Arg3 substitution and N-terminal 13-residue extension in IGF-1 LR3 reduce IGFBP affinity by approximately 1,000-fold, preventing this sequestration
Training adaptation (becoming stronger, faster, and more resilient) depends on cells that can absorb stress and respond to it
This process becomes significant for ATP synthesis through mitochondrial oxidative phosphorylation
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