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We therefore postulated that IGF-1R mAbs could represent a viable therapeutic tool to target IGF-1 action, and potentially mimic the beneficial effects associated with diminished IGF-1 signaling observed in animal models

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its activation can promote the accumulation of -catenin, thereby regulating the expression of downstream genes (Zhao et al., 2022b)

They represent the final date the manufacturer guarantees the full potency and safety of the medication when stored under ideal conditions

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