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To this end, we tested the expression Sirt1 and Foxo-1 in adipose tissues of the mice

Glutamine metabolism inhibition, using agents such as CB-839, has shown potential to suppress TCA cycle-mediated energy production and disrupt redox homeostasis, particularly in CRC subtypes resistant to 5-FU and oxaliplatin [145]

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It explicitly distinguishes these secondary mechanisms from any direct pharmacological effect of the medication itself