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All findings pertain strictly to laboratory and preclinical research contexts

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The major pathogenetic mechanism of GD is the stimulation of the thyroid stimulating hormone receptor (TSH-R) by autoantibodies that bind to it and promote thyrocyte proliferation and activity, resulting in thyroid hyperfunction (13)

Molinari C, Uberti F, Grossini E, Vacca G, Carda S, Invernizzi M, Cisari C