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The synergy between exogenous MOTS-c and exercise is not merely additive: because MOTS-c mimics one of the molecular consequences of high-intensity exercise (AMPK activation via AICAR accumulation), concurrent exercise training may amplify the peptide's effects on GLUT4 translocation and mitochondrial biogenesis
Exercise and the dipeptidylpeptidase IV inhibitor sitagliptin do not improve betacell function and glucose homeostasis in longlasting type 1 diabetesA randomised openlabel study
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