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Bump EA, Al-Sarraf R, Pierce SM, Coleman CN
27,28 Beyond mechanical stimuli, classical satiety hormones, such as cholecystokinin, augment both central GLP-1 neuronal activity and peripheral GLP-1 release
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The anti-inflammatory mechanisms appear to be independent of weight loss or glycemic improvement, suggesting direct immunomodulatory properties
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Heterologous expression of an engineered bifunctional bacterial GSH biosynthetic enzyme (GshF) in mitochondria enables mitochondrial GSH production and ameliorates the metabolic and proliferative defects caused by its depletion
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