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The Journal of Laboratory and Clinical Medicine
Mori, K., Nagao, H
2.2 Neuroinflammation in the brain and cognitive dysfunction in diabetes Neuroinflammation itself is a defence mechanism against acute the CNS damage and ameliorates the effects of toxic substances produced in brain nerve cells, but persistent neuroinflammation inhibits nerve regeneration, leading to neurodegeneration making the patient cognitively impaired
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Finally, IGF-1 encourages differentiation of myoblasts, which is to say that it encourages the conversion of stem cell progeny from nonspecific pluripotent stem cells into dedicated muscle tissue
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