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The chemical structure of melatonin allows it to act as a trap for free radicals, the hyperproduction of which causes cell damage during ischemic brain damage (Srinivasan et al., 2015
This work was partly supported by the Tohoku Medical Megabank Project from MEXT, Japan Agency for Medical Research and Development (AMED
10.3390/ijms22042101 53 LeeJ.WooH.KangH.ParkY
Soufir, J
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it further discusses how diverse upstream pathologies converge on ferroptosis as a shared mechanism underlying cognitive dysfunction
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