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Accumulation of abasic sites induces genomic instability in normal human gastric epithelial cells during helicobacter pylori infection

Additionally, S14, S17, and S18 of GLP-1 interact with the residues of ECL2 like C 296 , W 297 , T 298 , R 299 and N 300 , whereas no such interactions are observed for SR18

Despite the presence of DPP-4 inhibitor we were able to detect cleavage of both monotreme GLP-1 peptides (Supplementary Fig

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