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Hepatic steatosis is a consequence of lipid acquisition exceeding lipid disposal, for example, fatty acid uptake and de novo lipogenesis surpassing lipid export and fatty acid oxidation ( de novo lipogenesis, such as acetyl-Coenzyme A carboxylase ( Acaca ) and fatty acid synthetase ( Fasn ), and fatty acid uptake gene CD36 were increased by feeding a MCD diet for 3 weeks (Figures 2G-I)

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This GILZ homology is also proposed to underlie observed links between DSIP expression and adipogenesis, metabolic syndrome, and the transcriptional response to glucocorticoid challenge