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J Neurosci 34(15):53225334 Holst JJ (2007) The physiology of glucagon-like peptide 1

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These mechanisms are not fully understood but include central nervous system carbohydrate reduction and glycolysis inhibition, changes in neuronal excitability mediated by ATP-sensitive potassium channels through alterations in mitochondrial function, inhibition of the mammalian target of rapamycin (mTOR) pathway, and inhibition of glutamatergic excitatory synaptic transmission [72]

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