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In endothelial cell models, BPC-157 significantly enhanced ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formation

For a patient population already managing stress physiology and body composition, avoiding unnecessary cortisol elevation matters

It has been suggested that decreased activity of G6PD in the lenses reduces glutathione activity, making the lens more susceptible to oxidative stress [17]

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