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Deng-Bryant et al., 2011)

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The detection of HSMGSH in cells not exposed to exogenous FA indicates that cellular metabolism continuously produces sufficient FA to react with GSH yielding HSMGSH (Fig

we also determined whether the possible underlying mechanisms of its effects were associated with Nrf2-regulated anti-oxidative, mTOR-mediated anti-apoptotic, and/or autophagy systems

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This will involve multi-omics integration and advanced metabolic imaging to comprehensively analyze the sulfur/selenium metabolism, energy metabolism, and redox networks in which SQOR is embedded