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Copper-induced cell death and the protective role of glutathione: the implication of impaired protein folding rather than oxidative stress
After a host is infected with a pathogen, the peripheral blood copper level is significantly increased to meet the need to synthesize cytokines such as interleukin 2 to enable the body to achieve an efficient immune response [202]
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Endoplasmic reticulum-resident selenoproteins as regulators of calcium signaling and homeostasis
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