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n = 8), while a second group received 5A-1MQ treatment at 32 mg/kg of active pharmaceutical ingredient (injections of 4 mg 5A-1MQ monochloride salt/mL sterile saline dosed at 10 mL/kg body weight) after being switched from WD to LD (termed the WD/LD-T group
Diabetologia 60 , 296305 (2017)
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[3] While human trials remain limited, these findings raise interest in possible therapeutic applications
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[46] [47] [48] Moreover, it was further indicated that the formation of antibodies to gastric cells was autosomal dominant gene determined, and the presence of antibodies to the gastric cells might not be necessarily related to the occurrence of atrophic gastritis related to PA
