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[40] It contributes to metabolic flexibility, [41] and helps keep glucose levels down in the body while improving insulin sensitivity
117 UA-induced inflammation leads to the recruitment and activation of effector T cells at the site of crystal deposition, thereby exacerbating local inflammation
Oxidative stress is controlled very tightly in the thyroid gland via the synthesis of glutathione peroxidase, which can safely inactivate sources of oxidative stress, such as hydrogen peroxide and free radicals
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Disruption of the ROS-AMPK-Nrf2 axis has been implicated in muscle atrophy associated with disuse, ischemia-reperfusion injury, and metabolic stress (Coughlan et al., 2016)
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