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Adipose tissue converts androgens to oestrogens via the enzyme aromatase

To our best knowledge, this is the first systematic review to address these gaps by synthesizing evidence in CAD patients across mechanistic markers such as oxidative stress, inflammatory mediators, autophagy-related markers and post-PCI myocardial-injury biomarkers together with clinical endpoints such as ventricular arrythmia, mortality and angina

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215 In addition, mitochondrial STAT3 limits excessive ROS production, thereby curbing ROS-induced mitophagy

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