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In contrast, LCA primarily inhibits apoptosis in damaged cells by disrupting mitochondrial function and mimics growth factors to activate the Wnt/-catenin pathway, thereby maintaining stem cell characteristics and driving aberrant proliferation (Farhana et al., 2016) (Figure 2A)

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It was shown that the deletion of E155 in GSTO1-1 has a significant effect on enzymatic stability and may consequently alter the physiologic role of the enzyme in the brain regions 53

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