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One cause (among several) of interstitial cystitis is overactive mast cells in the bladder

Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity

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2,3,4 Loss-of-function mutations in NF1 promote tumor proliferation, survival, and therapeutic resistance, and have emerged as poor-prognosis genomic alterations across multiple solid tumors, including BC