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These models permit the testing not only of thyroid hormone replacement but also act as a vehicle to test putative interventions for developmental deficits due to thyroid hormone insufficiency (Kim et al

Although, the molecular mechanisms responsible for the anti-inflammatory effects of GLP-1/GLP-1R signals are still under investigation, the PKA/CREB pathway axis and associated active components may be major signals responsible for the anti-inflammatory effects of GLP-1/GLP-1R [23]