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Consequently, it is of major clinical significance to develop a biomaterial capable of inhibiting ferroptosis and inflammation of degenerative NPCs via reducing excessive ROS accumulation in the microenvironment, thereby restoring NPC viability and ECM homeostasis to promote disc repair, which would perform promising therapeutic efficacy on IDD

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Can GHK-CU cause copper toxicity

The significance of the accumulation of inosine stems from the fact that experiments have previously demonstrated that inosine promotes mitochondrial function in adipose tissue

[10] Admittedly, this is a smaller effect size, but the study duration was also shorter than what we've seen in Ozempic clinical trials

This review synthesizes current evidence regarding SGLT2 inhibitors and GLP-1 receptor agonists in DKD, examines their mechanisms and clinical outcomes, and highlights future research directions to address ongoing therapeutic needs