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Although glycine may lead to the formation of serine and subsequently D-serine, another NMDAR co-agonist, the levels of this amino acid are normal or low in NKH patients, suggesting that the levels of 5,10-methylene-THF, the cofactor for serine synthesis, are reduced [84, 85]

Ex-4 may also mitigate enzalutamide (ENZ) resistance and synergistically inhibit prostate cancer cell proliferation, potentially by inhibiting ENZ-induced Akt activation [69]

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