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Mutations in APP and presenilin genes can increase the A42/A40 ratio, accelerating plaque formation and downstream neurodegeneration, particularly in familial forms of AD [22]

Notably, the age-related increases in nitration of ECM-bound HGF were predominant on fast IIa and IIx myofibers 32 , corresponding with the fast fiber-preference observed in age-related muscle atrophy and impaired regeneration in humans in which fast IIb fibers are scarce 33,34,35, 39

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