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Strikingly, we find that the disordered FOXO4-DRI binds to the disordered p53 TAD2 and forms a transiently folded complex

HSCs, the primary producers of ECM, play an indispensable role in the pathogenesis of liver fibrosis (Hung et al., 2022)

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Research also examines its antioxidant activity through superoxide dismutase-like effects