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Its activity profile is best understood as systems-level: published in vitro and in vivo work has documented effects on fibroblast proliferation, collagen and growth-factor receptor expression, endothelial migration, nitric oxide system signalling, and dopamine and serotonin pathway markers

Baseline inflammatory markers, including C-reactive protein and interleukin-6, can provide an objective reference point for assessing systemic anti-inflammatory effects where that is a stated goal

Studies in aged non-human primates provide crucial translational evidence

Theres not a one-size-fits-all answer, and treatment for KP is usually a little bit slow going

Mouse model for molybdenum cofactor deficiency type B recapitulates the phenotype observed in molybdenum cofactor deficient patients