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Based on this, it is reasonable to conclude that the high E 2 environment associated with uterine fibroids can further compromise the antioxidant defense capacity of cells [Citation69]

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Another study found TMAO downregulates Piezo1, activating NOX4/ROS oxidative stress, causing abnormal calcium handling and fibrosis, constituting the molecular network of TMAO-induced cardiac fibrosis ( The above quadruple network links cardiac fibroblast proliferation, ER stress response, inflammatory signals and epigenetics, which together constitute the pathological basis of TMAO-induced fibrosis

Redox regulation of mitochondrial ATP synthase