Abstract Allergic asthma is characterized by type 2 inflammation and overnight worsening of symptoms, yet dynamic fluxes in cellular metabolic profiles driving time-of-day variation remain poorly defined
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Consequently, a deficiency in C4B protein in patients may impair its ability to bind microbial antigens
This suggested that both murine and human NCR + ILC3 could modulate tumor endothelial cell activation, promote leukocyte recruitment, favor the establishment of a pro-inflammatory microenvironment, and thus exert anti-tumoral functions
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Delgado-Peraza F, Nogueras-Ortiz CJ, Volpert O, et al