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So am I but here is the interesting bit

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Hence, GSH exhaustion based on multi-strategy therapy would be a desirable solution

The search parameters were set as follows: Trypsin/P was specified as the protease with up to 2 missed cleavages permitted

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Evidence from ischemic stroke models shows that HSP90 can activate both necroptosis and ferroptosis by promoting RIPK1 phosphorylation and inhibiting GPX4 activity, suggesting that activation of necroptosis may also potentiate ferroptosis [242]