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DHHC9-mediated GLUT1 S-palmitoylation promotes GBM tumorigenesis To investigate the functions of DHHC9-mediated GLUT1 S-palmitoylation in GBM development, we intracranially injected athymic nude mice with luciferase-expressing U87 cells with or without knockout of endogenous DHHC9 or GLUT1 and reconstituted expression of their WT counterparts, rDHHC9 C169S or rGLUT1 C207S (Fig

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doi: 10.1016/j.bbih.2020.100047 514 MaesMMihaylovaIKuberaMLeunisJ-CTwiskFNMGeffardM

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