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AMR makes infections harder to treat, increasing the risk of disease transmission, severe illness, and death (Murray et al., 2022)
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demonstrated that ASA-triggered 15-epi-LXA4 (ATL) and its metabolically stable analog ATL promoted the apoptosis of human neutrophils and facilitated the resolution of pulmonary inflammation by overriding the effect of myeloperoxidase (MPO) (Kebir et al., 2009)
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