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This bidirectional modulation suggests that BPC-157 acts as a homeostatic regulator of the NO system rather than simply an agonist or antagonist

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Large-scale gene expression analyses have suggested that GHK may regulate thousands of genes involved in tissue repair and inflammation control

This also leads to a potential answer to the unsettled discussion around elevated serum levels of copper in AD patients (Wang et al., 2021), and or concurrently through direct sequestration of copper in the serum that would exit through urinary excretion, as abnormal A levels and ratios are a common feature AD patients urine (Yang et al., 2021)

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