10.1016/j.jchf.2018.05.018 157 ScisciolaL.RizzoM
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ChaC1 overexpression can augment apoptosis by activating caspase-3/9, degradation of poly (ADP ribose) polymerase, induction of autophagy, ROS generation, increased intracellular calcium and loss of mitochondrial membrane potential (69)
Nicotinamide N-Methyltransferase Knockdown Protects against Diet-Induced Obesity
In the inflamed joint, multiple, functionally diverse macrophage subsets are now recognized ( Figure 2 Oxidative Phosphorylation in Inflammatory Effector Cells The major metabolic abnormality of RA CD4 + T cells lies in the mitochondria ( Figure 3 )
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