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The extracellular L-cysteine/L-cystine redox balance, and thus the synthesis of GSH, is finely regulated by the intracellular conversion of L-cystine into L-cysteine (10), and its impairment is related to oxidative stress and other pathological disorders

Oxidative stress injury is ubiquitous in the pathogenesis of AMS, HACE, and HAPE, and has become a hot topic of research in hypobaric hypoxic in recent years (Paul et al., 2018)

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Further, these precursors can be converted into other metabolites

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