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Consequently, SYK-deficient SCs accumulate dysfunctional mitochondria, leading to apoptosis and inadequate trophic support for regenerating axons
doi: 10.1038/sj.jid.5700421
These findings reveal a striking functional divergence: in microglia, SYK primarily drives phagocytosis [13, 15, 16], whereas in SCs, its paramount function centers on metabolic regulation
To verify the functionality of PAPCs, we re-clustered PAPCs with alveolar cells (Fig
Concurrently, immunofluorescence staining of axon density (TuJ1) showed a decrease in SYK-deficient nerves (Fig
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