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Musso, G., Gambino, R., Cassader, M., and Pagano, G

In pathological conditions such as cancer, dysregulation of these mechanisms leads to vascular hyper-permeability that in turn may exert direct effects on the tumor microenvironment including increased interstitial pressure and impaired therapeutic delivery [127]

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The accumulation of toxic lipids may cause sustained activation of JNK via ROS and MAP3K activation, while inhibition of JNK attenuates FFA-induced lipoapoptosis [69]