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These perturbations lead to the release of mtDAMPs, rewire immune cell metabolism toward pro-inflammatory or immunosuppressive phenotypes, and impair the bioenergetic capacity needed for effective host defense and tissue repair ( Figure 2 )

doi: 10.1080/19490976.2025.2517377 580 PanSHsuJCHungKTHoCJ

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Environmental exposure, DNA methylation, and gene regulation

TCF4 knockdown can promote ferroptosis and increase the cisplatin sensitivity of gastric cancer cells, 197 suggesting that abnormal activation of Wnt/-catenin pathway increases ferroptosis resistance in gastric cancer cells, offering a possible therapeutic strategy for enhancing chemotherapy sensitivity in patients with advanced gastric cancer