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Kasper Jacobsen Kyng, MD, PhD, neonatologist, Aarhus University Hospital, Department of Pediatrics and Adolescent Medicine, Aarhus, Denmark

GCL is specific for the glutamyl moiety and is regulated physiologically by: (a) non-allosteric feedback competitive inhibition by GSH (Ki=2.3mM), which involves binding of GSH to the glutamate and another site on the enzyme [8, 9] and (b) availability of its precursor, cysteine [1] .The apparent Km values of GCL for glutamate is 1.8 mM, and for cysteine and 0.10.3 mM [9]

Following liver resection, elevated bile salts activate FXR and upregulate IL-6/STAT3/c-JUN signaling to promote regeneration

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doi: 10.1186/s12935-025-03944-2

A few possible mechanisms have been proposed to explain the link between vitamin K and blood sugar metabolism