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In triple-negative breast cancer (TNBC) cells treated with isorhamnetin (IH) and chloroquine (CQ), ROS induced Drp1-dependent mitochondrial fission and apoptosis by mediating the activation and mitochondrial translocation of CaMKII

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Bone marrow stromal cells modulate sensitivity of CXCR4-deficient CML cells to pharmacological inhibitors of PI3K/AKT signaling

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