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And GAS could block the mitogen-activated protein kinases (MAPK) signal pathway, and suppress the inflammatory mediators such as cyclic adenosine monophosphate (cAMP)-response element binding protein (CREB) and nuclear factor kappa-B (NF-B) phosphorylation, in PTZ-treated epileptic mice [35, 36]

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Lack of organelles and therefore the mitochondria-associated apoptotic machinery has raised concerns that highly specialized terminally differentiated RBCs can undergo apoptosis

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