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Second, given that the activation of GSR and TXNRD1 are involved in ROS elimination, if the LCS3-triggered lethality of VRK2-KO PC cells could be attributed to inhibiting the activity of both GSR and TXNRD1, then the level of ROS generated by LCS3 should have been much higher than those produced by the treatment of the TXNRD1 inhibitor auranofin

This is achieved by the ability of erythrocytes to scavenge and detoxify exogenous neutrophils-, monocytes- or endothelial cells-derived reactive oxygen species (ROS) and reactive nitrogen species (RNS) [53, 54]

Lescaudron, L., Creuzet, S.E., Li, Z., Paulin, D

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Because the treatments (V, CS, C, S, WM, PF) were not distributed evenly among the experiments, experiment was not used as a covariate in the pooled analysis