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Tirzepatide is a dual glucose-dependent insulinotropic polypeptide (GIP) and GLP-1 receptor agonist, not a pure GLP-1 agonist
In practice, this makes complete sense, as we naturally form an early hypothesis and then refine and test that hypothesis through the observation and testing stage before drawing a conclusion (the diagnosis)
At each dose increase: Expect 1 to 2 weeks of increased GI sensitivity
[9] [10] Simultaneously, tirzepatide suppresses glucagon secretion, slows gastric emptying, and promotes satiety through central nervous system pathways
