One exception is the analysis of fluorescent protein substrates such as rxYFP or roGFP2, which allow a direct continuous detection of the reversible glutaredoxin-dependent protein disulfide reduction and formation in vitro 14,15,31,32,33,34,35
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Pathophysiologically, it manifests as root deficiency ( Ben Xu ) with branch excess ( Biao Shi ), predominantly presenting as interior excess-heat syndrome ( Li Shi Re Zheng ) (Mao et al., 2003)
However, human research remains mixed
