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PMID: 41506148

Rathore SS, Hinn AR, Cooper LS, Tyroler HA, Rosamond WD

Together, these data demonstrate a unique role for ILC2s in both initiating allergic airways disease and in responding to cues such as melatonin to dampen their effector phenotypes

Because these are time-dependent events, it is critical to always assess multiple time points in a pathophysiology and use parameters specific for apoptosis, while keeping in mind that many apoptosis parameters can also be modulated during a necrotic cell death

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[1] In order for the free fatty acids to become oxidized within the mitochondria to produce energy in the form of ATP (adenosine triphosphate) , they need some assistance