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Hyperphagia and increased fat accumulation in two models of chronic CNS glucagon-like peptide-1 loss of function

Dietary oxidative stress and antioxidant defense with an emphasis on plant extract administration

First, a committee recommendation is advice: the panel cannot change the list, and FDA is free to disagree

Glucagon receptor activation appears counterintuitive in obesity and diabetes therapy because glucagon acutely increases hepatic glucose output

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This observation led to the development of (Pro3)GIP, an analog of GIP with a glutamic-acid-to-proline substitution at position 3, improving glucose homeostasis and preventing weight gain in animal models of diabetes and obesity (Sparre-Ulrich et al., 2016)