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There are hints that constitutive activation of PI3KAKT signaling could be exploited therapeutically because it underlies a differential stress response between malign and benign cells, such that only the latter increase their resistance against cytotoxic insults upon reduction of growth factors induced by CR

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In the brainstem, it activates neurons in the area postrema and NTS that relay visceral fullness signals

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Decreased stamina, loss of muscle mass, and weight gain may also occur