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The mechanisms behind the renoprotective effects of GLP-1 RAs involve anti-albuminuric, anti-inflammatory, natriuresis, anti-atherogenic, and anti-oxidant effects through protein kinase A signaling, induction of natriuresis by inhibiting sodium-hydrogen exchanger 3, and a reduction in hyperfiltration 27,30,31

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demonstrated that cold atmospheric plasma treatment can induce the production of NO, which further stimulates the expression of VEGF and SDF-1 at the wound site, promotes the recruitment of mesenchymal cells to the wound area and facilitates the formation of new blood vessels [122]

*Correspondence: Yan Luo, [email protected]

J Biol Chem 273:2332723334